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n-butylidenephthalide protects against dopaminergic neuron degeneration and α-synuclein accumulation in Caenorhabditis elegans models of Parkinson's disease.

机译：正丁基苯酞在帕金森氏病的秀丽隐杆线虫模型中防止多巴胺能神经元变性和α-突触核蛋白积聚。

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Ru-Huei Fu; Horng-Jyh Harn; Shih-Ping Liu; Chang-Shi Chen; Wen-Lin Chang; Yue-Mi Chen; Jing-En Huang; Rong-Jhu Li; Sung-Yu Tsai; Huey-Shan Hung; Woei-Cherng Shyu; Shinn-Zong Lin; Yu-Chi Wang;
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1. LRRK2-mediated neurodegeneration and dysfunction of dopaminergic neurons in a Caenorhabditis elegans model of Parkinson's disease. [J] . Yao C, El Khoury R, Wang W, Neurobiology of disease . 2010,第1期

机译：在帕金森氏病秀丽隐杆线虫模型中，LRRK2介导的神经变性和多巴胺能神经元功能障碍。
2. Caffeine reduces deficits in mechanosensation and locomotion induced by L -DOPA and protects dopaminergic neurons in a transgenic Caenorhabditis elegans model of Parkinson’s disease [J] . Rafael Vincent M. Manalo, Paul Mark B. Medina Pharmaceutical Biology . 2020,第1期

机译：咖啡因减少了L -Dopa诱导的机械淤积和运动的缺陷，并保护多巴胺能神经元在帕金森病的转基因Caenorhabdise秀丽叶子中
3. Caenorhabditis elegans as a model system for identifying effectors of α-synuclein misfolding and dopaminergic cell death associated with Parkinson's disease [J] . HarringtonA.J., KnightA.L., CaldwellG.A., Methods: A Companion to Methods in Enzymology . 2011,第3期

机译：秀丽隐杆线虫作为识别与帕金森氏病相关的α-突触核蛋白错误折叠和多巴胺能细胞死亡的效应子的模型系统
4. Identification of novel alpha-synuclein post-translational modifications isolated from MAO-B over-expressing models of Parkinson's disease. [C] . Danielson SR, Held JM, Kumar J, ASMS Conference on Mass Spectrometry and Allied Topics . 2008

机译：鉴定帕金森病的Mao-B杂交模型中分离的新型α-突触核蛋白的翻译后修饰。
5. Molecular mechanisms of PKCdelta in neurotoxin-induced apoptotic death of nigral dopaminergic neurons: Relevance to the pathogenesis of Parkinson's disease. [D] . Yang, Yongjie. 2005

机译：神经毒素诱导的多巴胺能神经元凋亡死亡中PKCdelta的分子机制：与帕金森氏病的发病机制有关。
6. n-Butylidenephthalide Protects against Dopaminergic Neuron Degeneration and α-Synuclein Accumulation in Caenorhabditis elegans Models of Parkinsons Disease [O] . Ru-Huei Fu, Horng-Jyh Harn, Shih-Ping Liu, -1

机译：正丁烯酞可预防帕金森氏病秀丽隐杆线虫模型中的多巴胺能神经元变性和α-突触核蛋白积累
7. Caffeine reduces deficits in mechanosensation and locomotion induced by L-DOPA and protects dopaminergic neurons in a transgenic Caenorhabditis elegans model of Parkinson’s disease [O] . Rafael Vincent M. Manalo, Paul Mark B. Medina 2020

机译：咖啡因减少了L-DOPA诱导的机械化和运动的缺陷，并保护多巴胺能神经元在帕金森病的转基因Caenorhabdisegans模型中保护多巴胺能神经元

n-butylidenephthalide protects against dopaminergic neuron degeneration and α-synuclein accumulation in Caenorhabditis elegans models of Parkinson's disease.

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